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cholera toxin beta subunit band brain
(4 results)?
Anti-inflammatory effect of cholera toxin B subunit in experimental stroke.
J Neuroinflammation. 2016 Jun 13;13(1):147. doi: 10.1186/s12974-016-0610-y.
J Neuroinflammation. 2016.
PMID: 27296014
Free PMC article.
BACKGROUND: Cholera toxin B subunit (CTB) has multifaceted immunoregulatory functions. ...Cytokines, microglia/macrophage activation, and transcriptional factors in the ischemic brain were tested. The mRNA expressions of IL-1beta and TNF-alpha were tes …
BACKGROUND: Cholera toxin B subunit (CTB) has multifaceted immunoregulatory functions. ...Cytokines, microglia/macropha …
A new method for simultaneous demonstration of anterograde and retrograde connections in the brain: co-injections of biotinylated dextran amine and the beta subunit of cholera toxin.
J Neurosci Methods. 1999 Sep 15;91(1-2):1-8. doi: 10.1016/s0165-0270(99)00055-2.
J Neurosci Methods. 1999.
PMID: 10522819
In studying reciprocally connected brain networks, it is advantageous to use techniques that allow simultaneous visualization of both efferent and afferent connections from a single injection site. We report on a new technique to achieve this using pressure injections of a …
In studying reciprocally connected brain networks, it is advantageous to use techniques that allow simultaneous visualization of both …
Cholera toxin and pertussis toxin on opioid- and alpha 2-mediated supraspinal analgesia in mice.
Life Sci. 1991;48(18):1721-7. doi: 10.1016/0024-3205(91)90208-s.
Life Sci. 1991.
PMID: 1850493
Cholera toxin, an agent that impairs the function of Gs transducer proteins, was injected (0.5 microgram/mouse, icv) and the antinociceptive activity of opioids and clonidine was studied 24h later in the tail-flick test. In these animals, an enhancement of the analg …
Cholera toxin, an agent that impairs the function of Gs transducer proteins, was injected (0.5 microgram/mouse, icv) and the a …
Oral administration of a fusion protein between the cholera toxin B subunit and the 42-amino acid isoform of amyloid-beta peptide produced in silkworm pupae protects against Alzheimer's disease in mice.
PLoS One. 2014 Dec 3;9(12):e113585. doi: 10.1371/journal.pone.0113585. eCollection 2014.
PLoS One. 2014.
PMID: 25469702
Free PMC article.
A key molecule in the pathogenesis of Alzheimer's disease (AD) is a 42-amino acid isoform of the amyloid-beta peptide (Abeta42), which is the most toxic element of senile plaques. In this study, to develop an edible, safe, low-cost vaccine for AD, a cholera toxin …
A key molecule in the pathogenesis of Alzheimer's disease (AD) is a 42-amino acid isoform of the amyloid-beta peptide (Abeta42), whic …
Guanine nucleotide dependent formation of a complex between choleragen (cholera toxin) a subunit and bovine brain ADP-ribosylation factor.
Biochemistry. 1991 Apr 16;30(15):3697-703. doi: 10.1021/bi00229a015.
Biochemistry. 1991.
PMID: 1901726
Cholera toxin activates adenylyl cyclase by catalyzing the ADP-ribosylation of Gs alpha, the stimulatory guanine nucleotide binding protein of the cyclase system. This toxin-catalyzed reaction, as well as the ADP-ribosylation of guanidino compounds and …
Cholera toxin activates adenylyl cyclase by catalyzing the ADP-ribosylation of Gs alpha, the stimulatory guanine nucleotide bi …
Comparison of the carbohydrate-binding specificities of cholera toxin and Escherichia coli heat-labile enterotoxins LTh-I, LT-IIa, and LT-IIb.
Infect Immun. 1988 Jul;56(7):1748-53. doi: 10.1128/iai.56.7.1748-1753.1988.
Infect Immun. 1988.
PMID: 3290106
Free PMC article.
The toxins form two antigenically distinct groups; members of each group cross-react but are not necessarily identical. Serogroup I includes cholera toxin (CT) and type I heat-labile enterotoxin (LT-I) of E. coli. ...LTh-I, like CT, probably binds to the terminal su …
The toxins form two antigenically distinct groups; members of each group cross-react but are not necessarily identical. Serogroup I includes …
Modulation of the humoral and cellular immune response in Abeta immunotherapy by the adjuvants monophosphoryl lipid A (MPL), cholera toxin B subunit (CTB) and E. coli enterotoxin LT(R192G).
Vaccine. 2005 Oct 25;23(44):5149-59. doi: 10.1016/j.vaccine.2005.06.018.
Vaccine. 2005.
PMID: 16054274
We compared the adjuvants monophosphoryl lipid A (MPL)/trehalose dicorynomycolate (TDM), cholera toxin B subunit (CTB) and Escherichia coli heat-labile enterotoxin LT(R192G) for their ability to induce a humoral and cellular immune reaction, using fibrillar A …
We compared the adjuvants monophosphoryl lipid A (MPL)/trehalose dicorynomycolate (TDM), cholera toxin B subunit (CTB) …
Nasal administration of cholera toxin B subunit-nerve growth factor improves the space learning and memory abilities in beta-amyloid protein(25-35)-induced amnesic mice.
Neuroscience. 2008 Jul 31;155(1):234-40. doi: 10.1016/j.neuroscience.2008.05.040. Epub 2008 Jun 5.
Neuroscience. 2008.
PMID: 18585441
Nerve growth factor (NGF) is a potential drug for Alzheimer's disease treatment, but delivering NGF to the brain is difficult. To increase the content of NGF in brain, we prepared cholera toxin B subunit (CB) -NGF by the improved sodium metaperi …
Nerve growth factor (NGF) is a potential drug for Alzheimer's disease treatment, but delivering NGF to the brain is difficult. To inc …
ADP-ribosylation by cholera toxin of membranes derived from brain modifies the interaction of adenylate cyclase with guanine nucleotides and NaF.
J Neurochem. 1988 Jun;50(6):1791-7. doi: 10.1111/j.1471-4159.1988.tb02480.x.
J Neurochem. 1988.
PMID: 2836559
We have developed a method to ADP-ribosylate the stimulatory guanine nucleotide-binding protein of adenylate cyclase (GS) in brain membranes by using cholera toxin. ...This method reveals that GS is a very abundant protein in membranes derived f …
We have developed a method to ADP-ribosylate the stimulatory guanine nucleotide-binding protein of adenylate cyclase (GS) in brain …
Mesoscopic Mapping of Visual Pathway in a Female 5XFAD Mouse Model of Alzheimer's Disease.
Cells. 2022 Dec 2;11(23):3901. doi: 10.3390/cells11233901.
Cells. 2022.
PMID: 36497159
Free PMC article.
Amyloid-beta (Abeta) deposition and Abeta-induced neurodegeneration appear in the retina and retinorecipient areas in the early stages of Alzheimer's disease (AD). ...Therefore, we investigated the alterations of visual circuits in the AD mouse model using anterograde trac …
Amyloid-beta (Abeta) deposition and Abeta-induced neurodegeneration appear in the retina and retinorecipient areas in the early stage …
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